What is Type 2 diabetes

Type 2 diabetes is ex-old-age diabetes. "Ex" because it used to occur in almost all cases in the elderly, but in recent years, the number of 30-year-old type 2 diabetics has increased by 70 percent. In addition, more and more children are now going through life with so-called old-age diabetes.

Type 2 diabetes develops very slowly over many years compared to type 1 diabetes, which often develops within a few weeks. In type 2 diabetics, the pancreas is still working very well.

The beta cells produce insulin and everything would be fine if there were not the so-called insulin resistance of the body cells.

This means that the cells in the body of a type 2 diabetic (especially muscle, liver, and fat cells) no longer seem to recognize the insulin or only difficult to recognize and therefore no longer allow the glucose to enter the cell interior.

The sugar remains in the blood, although the cells are in urgent need of it and therefore repeatedly emit "we need glucose" signals. As a result, the pancreas produces more and more insulin and yet the amount produced never seems to be enough.

The blood sugar level remains elevated because the cells absorb only insufficient glucose.

The type 2 diabetic does not suffer from insulin deficiency. Although the pancreas can exhaust itself during the course of the disease and actually stop its service, this only happens in the case of type 2 diabetics in the final stage and in most cases only when the patient does not make friends with a healthier diet and lifestyle could.

Type 2 diabetes: conventional medicine prescribed antidiabetics

Thus, for the time being, insulin will not be given to type 2 diabetes (as long as the pancreas is still working). Type 2 diabetics receive instead of so-called antidiabetics from their orthodox therapist.

These are drugs that inhibit, for example, the release of glucose from the liver or block the glucose uptake from the intestine.

Other antidiabetics are said to increase the effect of the body's own insulin. In this way, the increase in blood sugar levels should be stopped with great force.

Often, such drugs were also administered, which stimulated the pancreas to even higher insulin production, but it has now been found that these drugs lead to an even faster depletion of the pancreas and therefore end so that the patient must inject insulin.

Type 2 Diabetes: The Symptoms

While type 1 diabetes is relatively quickly and clearly noticeable (high thirst, frequent urination, fatigue, etc.), incipient type 2 diabetes is often inconspicuous, as the pancreas may initially encounter increasing insulin resistance or increased insulin production ,

Although blood sugar levels can still rise, most people do not notice their onset of type 2 diabetes at this early stage.

Only when the pancreas becomes tired after mostly years of excessive insulin production and can no longer deliver enough insulin to the blood, the blood sugar level rises noticeably for the person affected, which can be shown in fatigue and loss of performance.

Type 2 Diabetes: The consequences

A permanently elevated blood sugar level leads to long-term damage to the body. The first victims are the small and large blood vessels. Their walls are thickened by the chronically elevated blood sugar levels and lose their elasticity.

It can also lead to tissue growth within the blood vessels, which does not improve their permeability. At the same time, a high blood sugar level affects the platelets (platelets). The result is an increased coagulation tendency, ie thicker blood.

Unfortunately, the combination of thickened blood vessel walls and viscous blood is anything but harmless. In the large blood vessels, therefore, diabetics have arteriosclerosis much faster than non-diabetics.

Possible consequences of these blood vessel changes are:

• ·Heart attack
•  Stroke
•   kidney failure
• Eye problems (green or cataract, the danger of blindness due to damage to the retinal vessels)
• Nervous problems (eg diabetic polyneuropathy: damage to the blood vessels that supply the nerves, which can develop in calf cramps, nerve pain or even paralysis)
•  Circulatory disorders that can manifest in the so-called diabetic foot. In the case of the diabetic foot, the small vessels in the foot in the toe or heel area are so damaged that tissue can die off at these points.

Type 2 Diabetes: The Possible Causes

The type 2 diabetic is even officially considered a disease that can develop due to an unhealthy diet and lifestyle. Consequently, conventional medical therapists are now recommending their patients to make three fundamental lifestyle changes:

Eat less sugar and carbohydrates, reduce body weight and regular exercise. In detail, the possible causes of type 2 diabetes are the following:

1. wrong diet:

Incorrect diets based on large amounts of carbohydrate-containing foods and sweetened drinks combined with lack of exercise (see next point) in many people over a number of years eventually lead to the insulin resistance of the cells described.

One has to imagine the following: A potential type 2 diabetic eats (in most cases) preferably bread, rolls, jam, pasta, white rice, cakes, biscuits, chocolate, ice cream, sweet snacks, sweetened milk products, and drinks sugared Soft drinks or sweetened coffee or tea.

These large quantities of sugar, starch, and white flour cause an above-average increase in blood sugar levels several times a day. As a result, the pancreas must repeatedly provide huge amounts of insulin to deliver the tidal wave of sugar from the blood into the cells.

However, a potential type 2 diabetic usually does not move much at the same time and thus needs little energy. So his cells do not need to produce much energy. If they have little energy to produce, then they need little fuel (sugar).

As a result, the organism suffers from an extreme sugar surplus with extremely low sugar requirements. Since the cells do not need the sugar offered by the insulin in excess, they sometimes really dull. They do not respond to insulin anymore. They are insulin resistant.

2. lack of exercise

Physical inactivity is also a factor that promotes the development of insulin resistance and therefore diabetes.

3. Overweight:

Obesity is considered a risk factor for the development of insulin resistance. Studies have shown that 65-70 percent of all severely overweight individuals are more or less insulin resistant.

4. Chronic inflammations:

Chronic inflammations are now considered as possible triggers for many different diseases, such. As for atherosclerosis, but also for diabetes mellitus. According to some scientists, it is precisely the above-mentioned obesity that plays a role, especially when a thick BELLY is present.

In the abdominal fat should often be unnoticed sources of inflammation, which can lead to the insulin that can no longer fulfill its function.

Obviously, fat on the thighs or other parts of the body should not affect this type of diabetes. Chronic inflammation can also lead directly to damage to insulin-producing beta cells in the islets of Langerhans.

5. fatty liver:

An existing fatty liver can promote the development of diabetes. A fatty liver, in turn, is created with preference in alcohol abuse and a general tendency to masslessness (too much sugar, too much fat, too much fast food, etc.), so that even in diabetes excessive alcohol consumption can cause an exacerbation.

6. Antioxidant deficiency:

An antioxidant deficiency is often underestimated but can favor or worsen almost any disease - and thus diabetes. An antioxidant deficiency occurs as follows:

Free radicals destroy the insulin receptors * on the cell surface of the body cells and could, therefore, be responsible for the development of insulin resistance.

Because if the insulin receptors on the cell surface are no longer intact, then the insulin can no longer pass sugar into the cell, because the cell no longer recognizes the insulin as such and therefore no longer allows the sugar.

Antioxidants eliminate free radicals, preventing free radicals from damaging the insulin receptors and thus delaying the development of insulin resistance.

7. Magnesium deficiency:

Magnesium deficiency can contribute enormously to diabetes. Only an adequate level of magnesium in the blood will allow the pancreas to deliver enough insulin. Without magnesium, the pancreas can work only to a limited extent.

In addition, magnesium ensures that insulin can remove the sugar from the blood and transport it into the cells. If magnesium is missing, the cells make them dense and do not allow the glucose to enter or occur only rarely (incipient insulin resistance).

In studies (1), it has been shown not only that diabetics have a remarkably low magnesium level compared to healthy people, but also that the lower the magnesium level, the higher the inflammation markers in the blood.